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More research is needed to explore the link between the different genetic variants of G6PD and this alternate pathway in Heinz body catabolism that could possibly explain this hematological peculiarity

Instead, what I'm going to call these are category one peptides and category two peptides

These effects, when they occur, are usually short-lived

Defection of IFN--mediated mtDNA degradation leads to a high mitochondrial membrane potential in SLE monocytes, promoting their autoreactivity in a STING-dependent manner (76)